Liquid capsules The AquaRes technology links the oil molecules to the water molecules through a relatively weak hydrogene-bridge bond in the stomach. So in case of physiological processes, there is no disorder in the assimilation of resveratrol which is a fat-soluble flavoniod. The special property of the system: considering the grade of dispersity it assimilates 100% without using the energies of the body. The antioxidant flavonoids in wine are from its red grape origins. Specifically, phytoalexin resveratrol from red grapes has been shown in cell studies to not only bolster immune defense, but also reduce tumor incidence and stall growth of many cancer cells. The original resveratrol molecules retain their structural properties throughout the technology. The quality of the materials and components used is conforms to the medical quality requirements. Resveratrol can be found in various products but their assimilation and utilization can only be effective, if the biliary acids physiologically present along with the pancreas secretions are available in the duodenum of 15-20 cm long, at the same time. In case the above conditions do not materialise, the vitamins and other ingredients will not be assimilated due to - e.g. various illnesses, mal-nutrition, digesting medicines’ effects, chemotherapy or radiation therapy of cancer patients, post-operation, weakened organism - causing disorders of the body. As usual, the two phases, fat and water, that originally never form a solution, are linked by a weak physical bond due to the surface-active emulsifier. This bond is a homogeneous disperse system with relative stability. Advantages of AquaRes: Due to the chemical bond, the oil phase ensures the highest degree of dispersion in the water phase (in the stomach) This concentrate can be deluted without limitation. The original molecules retain their structural properties throughout the technology. In case of physiological processes, there is no disorder in the assimilation of fats and fat-soluble vitamins. In case of non-physiological conditions in the digestive system, assimilation of fat-soluble vitamins is not appropriate (current vitamin materials). The patented invention Dr. András Bertha was the first to have his invention patented under the title: ’Material for flat-soluble vitamins’ at the Hungarian Patents Office, registered under the number: P0002561 – PCT/HU01/00073. Physiological effects of Resveratrol Life extension The groups of Howitz and Sinclair reported in 2003 in the journal Nature that resveratrol significantly extends the lifespan of the yeast Saccharomyces cerevisiae, the worm Caenorhabditis elegans and the fruit fly Drosophila melanogaster. In 2006, Italian scientists obtained the first positive result of resveratrol supplementation in a vertebrate. Using a short-lived fish, Nothobranchius furzeri, with a median life span of nine weeks, they found that a maximal dose of resveratrol increased the median lifespan by 56%. Compared with the control fish at nine weeks, that is by the end of the latter's life, the fish supplemented with resveratrol showed significantly higher general swimming activity and better learning to avoid an unpleasant stimulus. The high fat diet was compounded by adding hydrogenated coconut oil to the standard diet; it provided 60% of energy from fat, and the mice on it consumed about 30% more calories than the mice on standard diet. Both the mice fed the standard diet and the high-fat diet plus 22 mg/kg resveratrol had a 30% lower risk of death than the mice on the high-fat diet. Gene expression analysis indicated the addition of resveratrol opposed the alteration of 144 out of 155 gene pathways changed by the high-fat diet. Insulin and glucose levels in mice on the high-fat+resveratrol diet were closer to the mice on standard diet than to the mice on the high-fat diet. However, addition of resveratrol to the high-fat diet did not change the levels of free fatty acids and cholesterol, which were much higher than in the mice on standard diet. Cancer prevention In vitro resveratrol interacts with multiple molecular and has positive effects on the cells of breast, skin, gastric, colon, esophageal, prostate, and pancreatic cancer, and leukemia.However, the study of pharmacokinetics of resveratrol in humans concluded that even high doses of resveratrol might be insufficient to achieve resveratrol concentrations required for the systemic prevention of cancer. This is consistent with the results from the animal cancer models, which indicate that the in vivo effectiveness of resveratrol is limited by its poor systemic bioavailability. Accordingly is so important the water-soluble form and the highest bioavailability. The strongest evidence of anti-cancer action of resveratrol exists for tumors it can come into direct contact with, such as skin and gastrointestinal tract tumors. For other cancers, the evidence is equivocal, even if massive doses of resveratrol are used. Thus, topical application of resveratrol in mice, both before and after the UVB exposure, inhibited the skin damage and decreased skin cancer incidence. However, oral resveratrol was ineffective in treating mice inoculated with melanoma cells. Resveratrol given orally also had no effect on leukemia and lung cancer; however, injected intraperitoneally, 2.5 or 10 mg/kg of resveratrol slowed the growth of metastatic Lewis lung carcinomas in mice. Resveratrol (1 mg/kg orally) reduced the number and size of the esophageal tumors in rats treated with a carcinogen. In several studies, small doses (0.02-8 mg/kg) of resveratrol, given prophylactically, reduced or prevented the development of intestinal and colon tumors in rats given different carcinogens. Resveratrol treatment appeared to prevent the development of mammary tumors in animal models; however, it had no effect on the growth of existing tumors. Injected in high doses into mice, resveratrol slowed the growth of neuroblastomas. Athletic performance Johan Auwerx and coauthors published an online article in the journal Cell in November, 2006. Mice fed resveratrol for fifteen weeks had better treadmill endurance than controls. The study supported Sinclair's hypothesis that the effects of resveratrol are indeed due to the activation of the Sirtuin 1 gene. In a study of 123 Finnish adults, those born with certain increased variations of the SIRT1 gene had faster metabolisms, helping them to burn energy more efficiently—indicating that the same pathway shown in the lab mice works in humans. Neurodegenerative disease In November 2008, researchers at the Weill Medical College of Cornell University reported that dietary supplementation with resveratrol significantly reduced plaque formation in animal brains, the cause of Alzheimer and other Neurodegenerative diseases. In mice, oral resveratrol produced large reductions in brain plaque in the hypothalamus (-90%), striatum (-89%), and medial cortex (-48%) sections of the brain. In humans it is theorized that oral doses of resveratrol may reduce beta amyloid plaque associated with aging changes in the brain. Researchers theorize that one mechanism for plaque eradication is the ability of resveratrol to chelate (remove) copper.

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